If a mutation causes a cancer cell's growth factor receptor to continuously signal proliferation even without ligand binding, which consequence follows regarding programmed cell death pathways?

💡 Explanation

Continuous proliferation signaling from a mutated growth factor receptor leads to active suppression of apoptosis signals because the cell cycle checkpoints are bypassed. Therefore, apoptosis is inhibited, rather than increased or unaffected, due to unchecked growth signals overpowering the signals for programmed cell death.

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