If a novel drug prevents the reuptake of a specific neurotransmitter at a synapse in the brain, which consequence follows for signal transmission?

💡 Explanation

Preventing reuptake prolongs the neurotransmitter's presence in the synaptic cleft, leading to continued binding and prolonged signaling, because the neurotransmitter isn't cleared. Therefore, the signal is prolonged rather than terminated prematurely; receptor desensitization and neurotransmitter production halt would require different mechanisms.

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